You Searched For: N-Decanoyl-N-methyl-D-glucamine (MEGA-10)

Supplier: Merck

Description: Iridium(IV) oxide, Sigma-Aldrich®

Supplier: Merck

Description: Tin(IV) oxide, Sigma-Aldrich®

Catalog Number: (SIAL518131-10G)

Supplier: Merck

Description: Lead(IV) oxide, Sigma-Aldrich®

UOM: 1 * 10 g

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Supplier: Merck

Description: Titanium(IV) oxide rutile, Sigma-Aldrich®

Supplier: Merck

Description: Praseodymium(III,IV) oxide, Sigma-Aldrich®

Supplier: Merck

Description: Ruthenium(IV) oxide, Sigma-Aldrich®

Supplier: Merck

Description: Titanium(IV) oxide, Sigma-Aldrich®

Catalog Number: (SIAL253952-10G)

Supplier: Merck

Description: Terbium(III,IV) oxide, Sigma-Aldrich®

UOM: 1 * 10 g

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Supplier: Merck

Description: Germanium(IV) oxide, Sigma-Aldrich®

Supplier: Merck

Description: Ruthenium(IV) oxide hydrate, Sigma-Aldrich®

Catalog Number: (ROTH4479.1)

Supplier: Roth Carl

Description: Lead(IV) oxide

UOM: 1 * 100 g

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Catalog Number: (SIAL643009-100ML)

Supplier: Merck

Description: Cerium(IV) oxide nanoparticles 10%, colloidal dispersion in water <25 nm, Sigma-Aldrich®

UOM: 1 * 100 mL

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Catalog Number: (BOSSBS-0076R-A680)

Supplier: Bioss

Description: Functions as a cell surface receptor and performs physiological functions on the surface of neurons relevant to neurite growth, neuronal adhesion and axonogenesis. Involved in cell mobility and transcription regulation through protein-protein interactions. Can promote transcription activation through binding to APBB1-KAT5 and inhibits Notch signaling through interaction with Numb. Couples to apoptosis-inducing pathways such as those mediated by G(O) and JIP. Inhibits G(o) alpha ATPase activity (By similarity). Acts as a kinesin I membrane receptor, mediating the axonal transport of beta-secretase and presenilin 1. Involved in copper homeostasis/oxidative stress through copper ion reduction. In vitro, copper-metallated APP induces neuronal death directly or is potentiated through Cu(2+)-mediated low-density lipoprotein oxidation. Can regulate neurite outgrowth through binding to components of the extracellular matrix such as heparin and collagen I and IV. The splice isoforms that contain the BPTI domain possess protease inhibitor activity. Induces a AGER-dependent pathway that involves activation of p38 MAPK, resulting in internalisation of amyloid-beta peptide and leading to mitochondrial dysfunction in cultured cortical neurons. Provides Cu(2+) ions for GPC1 which are required for release of nitric oxide (NO) and subsequent degradation of the heparan sulfate chains on GPC1. Beta-amyloid peptides are lipophilic metal chelators with metal-reducing activity. Bind transient metals such as copper, zinc and iron. In vitro, can reduce Cu(2+) and Fe(3+) to Cu(+) and Fe(2+), respectively. Beta-amyloid 42 is a more effective reductant than beta-amyloid 40. Beta-amyloid peptides bind to lipoproteins and apolipoproteins E and J in the CSF and to HDL particles in plasma, inhibiting metal-catalysed oxidation of lipoproteins. Beta-APP42 may activate mononuclear phagocytes in the brain and elicit inflammatory responses.

UOM: 1 * 100 µl

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Supplier: Merck

Description: Vanadium(IV) oxide, Sigma-Aldrich®

Catalog Number: (SIAL643025-100ML)

Supplier: Merck

Description: Zirconium(IV) oxide, Sigma-Aldrich®

UOM: 1 * 100 mL

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Catalog Number: (SIAL643122-100ML)

Supplier: Merck

Description: Zirconium(IV) oxide, Sigma-Aldrich®

UOM: 1 * 100 mL

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